JIANGYONG MIN
Pharmacy in East Lansing, MI
License number
Michigan 5315037471
Issued Date
Jul 1, 2008
Expiration Date
Jun 30, 2013
Category
Pharmacy
Type
CS - 1
Address
Address
East Lansing, MI 48823
Organization information
See more information about JIANGYONG MIN at bizstanding.comJiangyong Min MD
A217 Clinical Ctr, East Lansing, MI 48824
Industry:
Neurologist
Phone:
(617) 877-3012 (Phone)
Jiangyong Min
Professional information
Dr. Jiangyong Min, East Lansing MI - MD (Doctor of Medicine)
Specialties:
Neurology
Address:
138 Service Rd, East Lansing 48824
(617) 877-3012 (Phone)
(617) 877-3012 (Phone)
Procedures:
Eeg (Electroencephalogram), Epilepsy, Lumbar Puncture, Multiple Sclerosis, Neuropathy, Stroke
Conditions:
Eeg, Epilepsy, Headache, Lumbar Puncture, Multiple Sclerosis (Ms), Neuropathy, Stroke
Awards:
Long-term improvement of cardiac function by transplantation of embryonic stem cells in post-infarcted rats, 2001
Languages:
English, Cantonese, Chinese, German, Mandarin
Physician At Michigan State University
Position:
Physician in Neurology at Michigan State University
Location:
East Lansing, Michigan
Industry:
Medical Practice
Work:
Michigan State University
since Jul 2007
-
Physician in Neurology
Jiangyong Min, East Lansing MI
Specialties:
Neurologist
Address:
138 Service Rd, East Lansing, MI 48824
Process For Inhibition Of Cerebral Damage Associated With Ischemia By Anthocyanins And Anthocyanidins
US Patent:
2013020, Aug 15, 2013
Filed:
May 10, 2012
Appl. No.:
13/468712
Inventors:
Muraleedharan G. Nair - Okemos MI, US
Jiangyong Min - East Lansing MI, US
Arshad Majid - Bradford, GB
Jiangyong Min - East Lansing MI, US
Arshad Majid - Bradford, GB
International Classification:
A61K 31/7048, A61P 7/02, A61K 38/49, A61K 31/352, A61P 25/00
US Classification:
424 9464, 514 27, 514456
Abstract:
A process is provided for inhibition of neural damage associated with an ischemic event that includes the administration of anthocyanin compound to a subject. After allowing sufficient time for the anthocyanin compound to reach the situs of the ischemic event, inhibition of neural damage associated with the ischemic event occurs. Neural damage is further inhibited by administration of the anthocyanin compound in conjunction with an agent effective in and specifically including tissue plasminogen activator. The anthocyanin compound in specific embodiments is in pure form or a mixture of two or more anthocyanins, or anthocyanidins or aglycones thereof, with the mixture being a natural product extract.A composition is also provided that includes ananthocyanin compound to cause blood clot dissolution. The agent being in specific embodiments is tissue plasminogen activator or aspirin.