DAVID SIGMUND LIEBESKIND, MD
Neurology in Los Angeles, CA
License number
California A62587
Category
Medical Practice
Type
Neurological Surgery
License number
California A62587
Category
Neurology
Type
Neurology
Address
Address
300 Medical Plz #B200, Los Angeles, CA 90095
Phone
(310) 794-1195
Personal information
See more information about DAVID SIGMUND LIEBESKIND at radaris.comName
Address
Phone
1016 Tiverton Ave, Los Angeles, CA 90024
745 Firth Ave, Los Angeles, CA 90049
745 Firth Ave, Los Angeles, CA 90049
Professional information
Dr. David S Liebeskind, Los Angeles CA - MD (Doctor of Medicine)
Specialties:
Vascular Neurology
Address:
UCLA Medical Center Neurology
300 Medical Plz SUITE B200, Los Angeles 90095
(310) 794-1195 (Phone)
300 Medical Plz SUITE B200, Los Angeles 90095
(310) 794-1195 (Phone)
Conditions:
Ataxia, Carotid Artery Disease, Cerebral Aneurysm, Cerebral Artery Thrombosis, Cerebral Vascular Disease, Cerebrovascular Disease, Diplopia, Epilepsy, Gait Abnormality, Low Back Pain, Myoclonus, Nystagmus, Parkinson's Disease, Sleep Apnea, Spinal Stenosis, Stroke, Transient Ischemic Attack (TIA), Vertigo, Visual Field Defects
Certifications:
Neurology, 2001, Vascular Neurology, 2005
Awards:
Healthgrades Honor Roll
Languages:
English
Education:
Medical School
New York University
Graduated: 1995
New York University
Graduated: 1995
David Sigmund Liebeskind, Los Angeles CA
Specialties:
Neurology, Vascular Neurology, Neurological Surgery
Work:
Ucla Stroke Ctr 1
710 Westwood Plz, Los Angeles, CA 90095 Ronald Reagan Ucla Medical Center
757 Westwood Plz, Los Angeles, CA 90095
710 Westwood Plz, Los Angeles, CA 90095 Ronald Reagan Ucla Medical Center
757 Westwood Plz, Los Angeles, CA 90095
Education:
New York University (1995)
Professor At Ucla
Position:
Professor at UCLA
Location:
Los Angeles, California
Industry:
Hospital & Health Care
Work:
UCLA
since 2004
-
Professor
Skills:
Science, Neurology, Clinical Research, Neuroscience, Research
David Sigmund Liebeskind, Los Angeles CA
Specialties:
Neurologist
Address:
710 Westwood Plz, Los Angeles, CA 90095
Board certifications:
American Board of Psychiatry and Neurology Certification in Neurology (Psychiatry and Neurology), American Board of Psychiatry and Neurology Sub-certificate in Vascular Neurology (Psychiatry and Neurology)
Noninvasive Method To Measure Intracranial And Effective Cerebral Outflow Pressure
US Patent:
2012013, May 31, 2012
Filed:
Feb 6, 2012
Appl. No.:
13/367304
Inventors:
Henrikas PRANEVICIUS - Kaunas, LT
Mindaugas PRANEVICIUS - Forest Hills NY, US
Osvaldas PRANEVICIUS - New York NY, US
David LIEBESKIND - Los Angeles CA, US
Mindaugas PRANEVICIUS - Forest Hills NY, US
Osvaldas PRANEVICIUS - New York NY, US
David LIEBESKIND - Los Angeles CA, US
International Classification:
A61B 5/055, A61B 8/06, A61B 5/021, A61B 5/02
US Classification:
600419, 600504, 600454, 600485
Abstract:
A system for detecting and measuring increased global or local intracranial pressure includes various devices for performing controlled occlusion of jugular cranial blood outflow and generating occlusion data related to said controlled occlusion, a cranial blood outflow pressure measurement device and a processor for processing jugular cranial blood outflow occlusion data and cranial blood outflow data to identify and/or measure a functional relationship between the jugular controlled occlusion and the jugular cranial blood outflow pressure. A device communicates the functional relationship a display device and/or a patient monitoring system. The processor also detects a state of equilibrium between the jugular cranial blood outflow pressure and the jugular occlusion pressure at occlusion. In yet another embodiment, intracranial and extracranial vessel compliance is measured by NIRS system, the equilibrated using external compression bladder, and equilibrium bladder pressure is displayed as local intracranial pressure.
Noninvasive Method To Measure Intracranial And Effective Cerebral Outflow Pressure
US Patent:
8109880, Feb 7, 2012
Filed:
Dec 12, 2007
Appl. No.:
11/954227
Inventors:
Osvaldas Pranevicius - New York NY, US
Mindaugas Pranevicius - New York NY, US
Henrikas Pranevicius - Kaunas, LT
Egidijus Marcinkevicius - Kaunas, LT
David S. Liebeskind - Los Angeles CA, US
Mindaugas Pranevicius - New York NY, US
Henrikas Pranevicius - Kaunas, LT
Egidijus Marcinkevicius - Kaunas, LT
David S. Liebeskind - Los Angeles CA, US
International Classification:
A61B 5/02, A61B 5/00
US Classification:
600490, 600500, 600504, 600506, 600561
Abstract:
A method to measure effective cerebral outflow pressure or intracranial pressure is disclosed. The craniospinal venous system has multiple anastomoses between the jugular veins and vertebral venous plexus. Jugular veins collapse with cervical compression or head elevation, when extrinsic pressure exceeds venous pressure. The vertebral venous plexus is exposed to intracranial pressure and collapses when intracranial pressure exceeds venous pressure. Vertebral venous plexus is not compressed with head elevation or cervical compression, because enclosure in the spinal canal protects veins from the direct effects of atmospheric pressure and cervical compression. Using cervical compression and/or head elevation blood outflow is redistributed between jugular veins and vertebral venous plexus, while the degree of cervical compression or head elevation indicates effective cerebral outflow pressure or ICP.
Venous Modulation Of Collateral Perfusion Of Cerebral Ischemia
US Patent:
2010031, Dec 16, 2010
Filed:
Oct 31, 2008
Appl. No.:
12/740623
Inventors:
Osvaldas Pranevicius - New York NY, US
Mindaugas Pranevicius - Forest Hills NY, US
David S. Liebeskind - Los Angeles CA, US
Mindaugas Pranevicius - Forest Hills NY, US
David S. Liebeskind - Los Angeles CA, US
Assignee:
The Regents of the University of California - Oakland CA
International Classification:
A61M 29/02
US Classification:
606194
Abstract:
A patient in whom blood diversion due to cerebral venous steal is present, and abolishment of the cerebral venous steal is indicated, is treated by increasing the cerebral venous pressure in the patient. This increase in cerebral venous pressure restores the collapsed cerebral vasculature, thereby increasing cerebral blood flow. The increase in cerebral venous pressure may be achieved using an occluding catheter in the superior vena cava or the internal jugular veins, using external compression of the cervical veins, or any other suitable mechanism. The occlusion may be controlled precisely during treatment, possibly as a function of cerebral blood flow, and after treatment the patient may experience a persistent effect because the cerebral vasculature is no longer collapsed.