DR. DAVID J CLARK, MD/PHD
Osteopathic Medicine at Miranda Ave, Palo Alto, CA
License number
California A65835
Category
Osteopathic Medicine
Type
Pain Medicine
Address
Address
3801 Miranda Ave, Palo Alto, CA 94304
Phone
(650) 493-5000
Professional information
David Clark - Palo Alto, CA
Work:
Institute of Astronomy, UNAM - Ensenada, B.C.
Resident Astronomer (2009 - 2014), Post Doctoral Researcher (2007 - 2009) Department of Astronomy, University of Florida - Gainesville, FL
PhD student
Resident Astronomer (2009 - 2014), Post Doctoral Researcher (2007 - 2009) Department of Astronomy, University of Florida - Gainesville, FL
PhD student
Education:
University of Florida
PhD in Astronomy University of Florida
MS in Astronomy Colby College
BA in Physics
PhD in Astronomy University of Florida
MS in Astronomy Colby College
BA in Physics
Skills:
Computer languages: Proficient in: IDL, Python, MySQL, PHP, HTML; class work with: Octave, R; Operating Systems: Mac OS, Linux, Windows XP; Tools and Software: Bash shell, Excel, Numbers, latex, Word, Pages, PowerPoint, Keynote; Methodologies: Designing and deploying/implementing databases in MySQL and writing web interface with PHP and HTML, writing scientific papers using Latex, class work in machine learning
Dr. David J Clark, Stanford CA - MD (Doctor of Medicine)
Specialties:
Pain Medicine
Address:
Stanford Office
300 Pasteur Dr, Stanford 94305
(650) 723-4000 (Phone)
300 Pasteur Dr, Stanford 94305
(650) 723-4000 (Phone)
Certifications:
Anesthesiology, 1997, Pain Medicine, 2009
Awards:
Healthgrades Honor Roll
Languages:
English
Hospitals:
Stanford Office
300 Pasteur Dr, Stanford 94305
Stanford Hospital and Clinics
300 Pasteur Dr, Stanford 94305
300 Pasteur Dr, Stanford 94305
Stanford Hospital and Clinics
300 Pasteur Dr, Stanford 94305
Education:
Medical School
Vanderbilt University School Of Medicine
Graduated: 1992
University Of Wa School Of Med
Vanderbilt University Med Center
Vanderbilt University School Of Medicine
Graduated: 1992
University Of Wa School Of Med
Vanderbilt University Med Center
David John Clark, Palo Alto CA
Specialties:
Anesthesiology, Pain Medicine, Internal Medicine, Cardiovascular Disease, Pain Medicine
Work:
Palo Alto Veterans Affairs Medical Center
3801 Miranda Ave, Palo Alto, CA 94304
3801 Miranda Ave, Palo Alto, CA 94304
Education:
Vanderbilt University (1992)
David John Clark, Palo Alto CA
Specialties:
Pain Management Specialist
Address:
3801 Miranda Ave, Palo Alto, CA 94304
Toxin Induced Sympathectomy
US Patent:
2007026, Nov 15, 2007
Filed:
Jan 26, 2005
Appl. No.:
10/587535
Inventors:
Ian Carroll - Mountain View CA, US
David Clark - Palo Alto CA, US
Sean Mackey - Los Altos CA, US
David Clark - Palo Alto CA, US
Sean Mackey - Los Altos CA, US
International Classification:
A61K 35/74, A61P 23/00, A61P 43/00, A61P 9/00, A61P 9/10
US Classification:
424780000
Abstract:
A selective and extended sympathetic nerve block is achieved by administration of a botulinum toxin at and/or around a targeted sympathetic ganglion. The toxin induced sympathetic block finds use for the treatment of sympathetically maintained pain. The toxin induced sympathetic block is also used to treat cardiovascular conditions of sympathetically maintained vasoconstriction; and of undesirable smooth muscle spasm.
Methods And Compositions For Treating Or Preventing Narcotic Withdrawal Symptoms
US Patent:
2010014, Jun 10, 2010
Filed:
Dec 4, 2009
Appl. No.:
12/631628
Inventors:
Gary PELTZ - Redwood City CA, US
David CLARK - Palo Alto CA, US
Lawrence CHU - Menlo Park CA, US
David CLARK - Palo Alto CA, US
Lawrence CHU - Menlo Park CA, US
Assignee:
The Board of Trustees of the Leland Stanford Junior University - Palo Alto CA
International Classification:
A61K 31/4178, A61K 31/444, A61K 31/485, A61K 31/495, A61P 25/36
US Classification:
51425504, 514397, 514296, 514282
Abstract:
This invention provides methods and pharmaceutical compositions for preventing or treating physical dependence and/or withdrawal associated with narcotic use, in particular by modulating a 5-HT3 receptor. Using a computational genetic approach in mice, a gene conserved between mice and humans was identified as candidate as a modulator of physical dependence to morphine. Administration of compounds that modulate 5-HT3 receptors was found to control withdrawal from morphine in mice and humans.